By Anitha Varghese MBBS BSc MRCP, Dudley J. Pennell MD FRCP FACC
This name presents an simply digestible and conveyable synopsis of the method for you to go well with the wishes of cardiologists and cardiothoracic surgeons wishing to acquaint themselves with what CMR can do, and what it can't. starting with an summary of a few of the elemental rules of MRI, the subsequent chapters pay attention to the cardiac part of CMR with a later part on its extra validated vascular makes use of.
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Extra resources for Cardiovascular Magnetic Resonance Made Easy
B) Example of iron deposition in a patient with thalassaemia major. The IVS now has a dark epicardial rim indicating signiﬁcant cardiac iron loading (white arrow). Iron deposition within the liver is also heavy and the liver therefore appears black (white ellipse). This signal loss occurs because of disturbances in the relaxation parameters of the tissues brought about by the iron causing alterations in the local magnetic ﬁeld. (c) Another case of thalassaemia major which highlights the poor correlation between iron deposition in the heart (black arrow) and the liver (black ellipse).
Extrinsic contrast can be created by LGE, which uses an inversion recovery sequence and a gadolinium contrast agent (see Chapter 2). Gadolinium highlights areas of myocardium with expanded interstitium, such as ﬁbrosis, necrosis or inﬁltration. 6). MI leads to patterns of LGE which spread from the subendocardium outward to the subepicardium in the territory of a coronary artery. However, changes in myocarditis are the reverse and start at the subepicardium and can later become mid-myocardial, typically at the lateral wall.
AFD is an X-linked storage disease that causes left ventricular hypertrophy (LVH) and accounts for 1 to 3% of patients with phenotypic HCM. 7 Four-chamber SSFP cine CMR images from a 51-year-old man referred with an abnormal resting ECG but normal TTE and X-ray coronary angiography. There is (a) hypertrophy at the LV apex in diastole associated with (b) LV cavity obliteration in systole, indicating a diagnosis of apical HCM. 8 Post-contrast CMR study from a 43-year-old man with a strong family history of HCM, mildly decreased LV function on TTE, and normal coronary arteries.